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This push is part of a broader movement to consider sex as an important variable in biomedical research, to ensure that studies cover the range of possibilities rather than gleaning results from a single population. Results are starting to trickle out, and it’s clear that certain pain pathways vary considerably, with immune cells and hormones having key roles in differing responses. Today, inspired in part by Sorge and Mogil’s work and spurred on by funders, pain researchers are opening their eyes to the spectrum of responses across sexes. Others stuck with males because, well, that’s how things were done. At the time, many pain scientists worried that females’ hormone cycles would complicate results. By including male and female mice, they were going against the crowd.
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Sorge and Mogil would never have made their discovery if they had followed the conventions of most pain researchers. “We couldn’t figure out why.” Sorge and his adviser at McGill University, pain researcher Jeffrey Mogil, would go on to determine that this kind of pain hypersensitivity results from remarkably different pathways in male and female mice, with distinct immune-cell types contributing to discomfort 2. “It just didn’t work in the females,” recalls Sorge, now a behaviourist at the University of Alabama at Birmingham.
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The males behaved as the scientific literature said they would: they yanked their paws back from even the finest of threads.īut females remained stoic to Sorge’s gentle pokes and prods 1. To test for this response, Sorge poked the paws of mice using fine hairs, ones that wouldn’t ordinarily bother them. Robert Sorge was studying pain in mice in 2009, but he was the one who ended up with a headache.Īt McGill University in Montreal, Canada, Sorge was investigating how animals develop an extreme sensitivity to touch.
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